WormBase Tree Display for Interaction: WBInteraction000050587
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| WBInteraction000050587 | Interaction_type | Genetic | No_interaction | ||
|---|---|---|---|---|---|
| GI_module_three | Neutral | ||||
| Interactor | Interactor_overlapping_gene | WBGene00008681 | Interactor_type | Non_directional | |
| WBGene00013034 | Interactor_type | Non_directional | |||
| WBGene00014200 | Interactor_type | Non_directional | |||
| WBGene00015437 | Interactor_type | Non_directional | |||
| Variation_interactor | WBVar00249659 | Interactor_type | Non_directional | ||
| WBVar00249665 | Interactor_type | Non_directional | |||
| WBVar00249682 | Interactor_type | Non_directional | |||
| Interaction_summary | tat-1 RNAi alters phosphatidylserine (PS) distribution in the membrane of C. elegans germ cells so that PS staining was observed on the surface of many normal germ cells. In wild-type C. elegans, no PS staining was observed on the surface of normal germ cells. Similar amounts of germ cell PS staining were observed in scrm-4(tm624) scrm-(tm650), scrm-3(tm631) triple mutant animals treated with tat-1(RNAi) to that seen in wild-type animals treated with tat-1(RNAi). Thus, loss of phospholipid scramblases does not affect PS externalization in tat-1 deficient animals. | ||||
| Throughput | Low_throughput | ||||
| Interaction_RNAi | WBRNAi00077721 | ||||
| Interaction_phenotype | WBPhenotype:0001399 | ||||
| Paper | WBPaper00031830 |
