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WormBase Tree Display for Variation: WBVar00091501

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Name Class

WBVar00091501NamePublic_nameok192
Sequence_detailsSeqStatusPending_curation
Variation_typeAllele
OriginSpeciesCaenorhabditis elegans
LaboratoryRB
PersonWBPerson46
KO_consortium_allele
StatusLive
AffectsGeneWBGene00000393
InteractorWBInteraction000524394
DescriptionPhenotypeWBPhenotype:0000239Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Remarkcdf-1(ok192) mutant animals generated fewer P5.p - P7.p descendant nuclei than wild type animals (Table 2)Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
PenetranceIncomplete67% penetrantPaper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
EQ_annotationsAnatomy_termWBbt:0008590PATO:0000460Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
WBPhenotype:0000518Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Remarkcdf-1(ok192) mutants displayed a low penetrance of developmental defects under standard growth conditions.Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
PenetranceLowPaper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
WBPhenotype:0001749Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Remarkcfd-1(ok192) mutants exhibit zinc hypersensitivty (Figure 4)Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Affected_byMoleculeWBMol:00005064Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Phenotype_assayTreatmentEggs were placed on NGM agar plates with supplemental zinc sulfate (Figure 4A), and development was monitored daily; the percentage of animals that reached adulthood within four days is shown (Figure 4A). Each data point represents at least 100 eggs.Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Phenotype_not_observedWBPhenotype:0000593Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Affected_byMoleculeWBMol:00002862Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
WBPhenotype:0000640Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
RemarkIn an otherwise wild-type genetic background, the cdf-1(ok192) mutation did not cause gross defects in egg laying.Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
WBPhenotype:0000699Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
RemarkIn an otherwise wild-type genetic background, the cdf-1(ok192) mutation did not cause gross defects in vulval development.Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
EQ_annotationsAnatomy_termWBbt:0006748PATO:0000460Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
WBPhenotype:0001655Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Affected_byMoleculeWBMol:00003022Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
WBPhenotype:0002226Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Remarkcdf-1(ok192) mutants respond to cobalt like wild type animalsPaper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Affected_byMoleculeWBMol:00003058Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
Phenotype_assayTreatmentEggs were placed on NGM agar plates with supplemental cobalt chloride (Figure 4B), and development was monitored daily; the percentage of animals that reached adulthood within four days is shown (Figure 4B). Each data point represents at least 100 eggs.Paper_evidenceWBPaper00005255
Curator_confirmedWBPerson2987
ReferenceWBPaper00005255
MethodKO_consortium_allele