- systemic RNAi variant
Animals are defective for RNAi introduced by environmental means such as in the cases of RNAi triggered by soaking or feeding.
- autosomal nondisjunction meiosis
A non-sex chromosome fails to segregate away from its homologous chromosome, resulting in gametes with no or extra copies of the non-sex homolog.
- RNAi resistant
Animals show reduced susceptibility to gene silencing triggered by double stranded RNA (RNAi), compared to control.
- cytoplasmic RNAi defective
Animals fail to silence cytoplasmic-localized RNAs. Nuclear-localized RNAs may still be sensitive to RNAi.
- RNAi enhanced
Animals show increased susceptibility to gene silencing triggered by double stranded RNA (RNAi), compared to control.
- allophagy defective
Animals exhibit defects in allogenic (non-self) organelle autophagy, or allophagy, a macroautophagy process whereby organelles of non-self origin, like paternal mitochondria from sperm in a newly fertilized oocyte, are selectively degraded.
- allophagy abnormal
Animals exhibit variations in allogenic (non-self) organelle autophagy, or allophagy, a macroautophagy process whereby organelles of non-self origin, like paternal mitochondria from sperm in a newly fertilized oocyte, are selectively degraded.
- RNAi transport defective
Animals are deficient in the transport of silencing RNAs between cells during the systemic RNAi response process.
- RNAi response variant
Animals exhibit variations in their response to gene silencing triggered by double stranded RNA (RNAi), compared to control.