PDE axons failed to reach the ventral nerve cord or reached it at a position greater than approximately a 45 degree angle from the PDE cell body. The PDE axon guidance defects of wve-1 RNAi were increased from 5 percent to 50 in mig-2(mu28) animals.
When the rescuing GFP::WVE-1 transgene (pjIs1) was crossed into animals with null mutations in unc-40, sax-3 or vab-1, all three led to reduced accumulation of GFP::WVE-1 at the basolateral epidermis.
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