"expression of both the endogenous
ges-1 gene and an (integrated)
ges-1B transgene were examined in embryos that were homozygous for (apparently null) mutations in
lin-39 and in
mab-5, as well as in embryos that were homozygous for the deficiency nDf16, which removes the entire C. elegans homeotic cluster (Wood, 1988; Clark et al., 1993). The results are shown in Fig. 3. In all the mutant embryos examined, endogenous
ges-1 is expressed throughout the gut... In other words, we see no obvious requirement for homeotic genes in patterning of the C. elegans embryonic gut"