900+ Interactions found (0.009 seconds)

- Quercetin : daf-16 : kaempferol : myricetin : naringenin
- ttll-11 : ttll-15
- ttll-15 : ttll-5
- ced-10 : mig-2
- Interaction involving Cbr-lin-39
- tbg-1 : zyg-9
- ced-10 : mig-2

All tested flavonoids increased nuclear localization of DAF-16 in gut epithelial cells (myricetin p = 0.0469, quercetin p = 0.004, kaempferol p = 0.04 and naringenin p = 0.0455, Students t-test), demonstrating a reduced DAF-16 phosphorylation status after flavonoid treatment.

Deletion of individual TTLL enzymes does not impair viability or brood size, moreover microtubule morphology in the early embryo is unaffected (Chawla et al., 2016). The viability of the double mutants does not differ significantly from that of wild type in any case (Figure 1A, p>0.05; Students t-test). The brood size of the double mutants does not differ significantly from wild type with two exceptions: ttll-5 ttll-15 (p=0.04; Students t-test) and ttll-11; ttll-15 (p=0.01 Students t-test).

Deletion of individual TTLL enzymes does not impair viability or brood size, moreover microtubule morphology in the early embryo is unaffected (Chawla et al., 2016). The viability of the double mutants does not differ significantly from that of wild type in any case (Figure 1A, p>0.05; Students t-test). The brood size of the double mutants does not differ significantly from wild type with two exceptions: ttll-5 ttll-15 (p=0.04; Students t-test) and ttll-11; ttll-15 (p=0.01 Students t-test).

In most animals only P5.p or P7.p descendants showed migration defect, but in 45 percent both P5.p and P7.p descendants showed Mig defects.

The small nucleus phenotype of P7.p, P8.p, and P11.p was not suppressed by bh20 (data not shown).

"Interestingly, centrosomal ZYG-9 was significantly reduced in tbg-1(RNAi) embryos (Fig. 2, B and C), to ~30% (31.6 +/- 17.0%) of wild-type levels (P < 0.0001, t test)."

In most animals only P5.p or P7.p descendants showed migration defect, but in 45 percent both P5.p and P7.p descendants showed Mig defects. Many cells that should have divided along the transverse axis in P6.p and P7.p descendants instead divided along the longitudinal axis.

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