dsh-1 RNAi causes enhancement of the dsh-2 embryonic lethality. Embryonic lethality increased from 54.0 percent for dsh-2(RNAi) to 67.9 percent for RNAi of both dsh-1 and dsh-2.
The no intestine phenotype in dsh-2(RNAi),mig-5(RNAi),mom-2(ne141)animals is not as penetrant as mom-2(ne141)alone, but more penetrant than dsh-2(RNAi),mig-5(RNAi).
The END-3::GFP signal in Ea or Ep was significantly reduced in dsh-2(RNAi);mig-5(RNAi);hmp-2(RNAi) embryos com pared to untreated embryos and dsh-2(RNAi);mig-5(RNAi) embryos.
The no intestine phenotype in dsh-2(RNAi),mig-5(RNAi),mom-5(zu193) animals is not as penetrant as mom-5(zu193) alone, but more penetrant than dsh-2(RNAi),mig-5(RNAi).