"The
plx-2(0) mutation was unable to significantly enhance either the P cell midline alignment defect or the embryonic lethality of the
mab-20 null (Figures 5A and 5C). This is consistent with PLX-2 acting in the same pathway as MAB-20 to prevent these two defects and with the finding by Nakao et_al [7], that MAB-20 binds PLX-2."