Interaction data was extracted by a curator from sentences enriched by Textpresso. The interaction was attributed to the paper(s) from which it was extracted.
Animals exposed to both lon-2 and sma-4 RNAi only display the small phenotype, suggesting that sma-4 is epistatic to lon-2 (which normally causes a long phenotype)
Mutations in lon-2 (e678) enhanced HSN migration defects of sdn-1 (zh20) from 52% (n=111) to 77% (n=77; P,0.001). Similar enhancement of HSN migration defect was observed in combination with the sdn-1ok449 allele.